Besides that, we hypothesize that AD animals would show memory deficits and associated reductions in levels of GLUT3, GLUT1 and Cav1.2 while animals given early training would show persevered levels of GLUT3, GLUT1 and Cav1.2 compared to animals that are not given early training and are not under the memory loss spectrum. Here, SLC2A1 is linked to Alzheimer disease.