Moreover, ZIKV induced a shift of the monocyte population from a CD14+ CD16− toward a CD14+ CD16+, preferentially infecting them (Supplementary Fig. 2b, c), which correlates with studies that previously showed that the CD14+ CD16+ monocytic subset is particularly permissive to infection by other Asian ZIKV strains21,22. The gene discussed is CD14; the disease is infection.