Given that occasional monoallelic mutations in an N-terminal hotspot were suggested to confer FOXO1 insensitivity to AKT-dependent phosphorylation in BL, we included cell lines harboring both wild type (Ramos, BL-41, Daudi, Raji) and also mutated FOXO1 alleles (Namalwa, Jiyoye) (Table S1). This evidence concerns the gene AKT1 and Burkitt lymphoma.