Overexpression of miR‐210 in endothelial cells stimulates capillary‐like structure formation and VEGF‐driven cell migration, while down‐regulation of miR‐210 inhibits this effect.54, 55 Arif et al51 injected miR‐210 into the myocardium of a rat model of acute myocardial infarction, observed angiogenesis in cardiomyocytes after four weeks and found that the vascular density in the area around the infarct increased by a factor of two, indicating that angiogenesis was enhanced; there was also a twofold increase in VEGF expression levels. The gene discussed is VEGFA; the disease is myocardial infarction.