The data from the mouse models suggest that the inducible deletion of c‐Jun and JunB in HF‐SCs can initiate and maintain psoriasis‐like plaques in ear skin, the epidermal hyperplasia and inflammatory features of the disease, but it does not fully recapitulate the systemic effects observed in the DKO* psoriasis‐like mice. This evidence concerns the gene JUNB and hydrops fetalis.