Knock‐down of Foxn3 significantly decreased expression of Slc43a1, Oat, and Ass1, and increased expression of Hmgcs1 and Fbp1. As internal controls, Foxn3 and Myc transcripts were measured, and were significantly decreased and increased, respectively, confirming persistence of the AAV8 infection and the encoded shRNAs' effects. The gene discussed is FOXN3; the disease is infection.