RUBCN and metabolic dysfunction-associated steatotic liver disease: Knock down of Rubicon, an autophagy suppressor that is increased in NAFLD patients, via siRNA in HepG2 cells, BNL-CL2 cells, and murine primary hepatocytes attenuated autophagy impairment and reduced endoplasmic reticulum stress, apoptosis and lipid accumulation in NAFLD inducing conditions.