In humans, the aggregation-prone heterogeneous nuclear ribonucleoprotein (hnRNP) human fused in sarcoma (hFUS) could aberrantly form immunoreactive inclusion bodies in a range of neurological diseases classified as FUS-proteinopathies, and its homolog Drosophila FUS (dFUS) is a hsrω-interacting protein in Drosophila [14]. This evidence concerns the gene FUS and proteostasis deficiencies.