In experimental murine models of amyloidosis in vivo, IDOX specifically accumulated in amyloid deposits of amyloid A, immunoglobulin LC, transthyretin (TTR), amyloid β (Aβ) and β2microglobulin (β2M) but did not bind to the monomeric amyloid precursor [2]. This evidence concerns the gene HLA-G and amyloidosis.