Corilagin stimulated the expression and inhibited the degradation of inhibitor of κBα (IκBα), blocked nuclear factor-κB (NF-κB) activation, suppressed the stimulated p65 protein moving into the nucleus, and thus inhibited the NF-κB signaling pathway and promoted apoptosis in neural tumor cells [129]. The gene discussed is NFKB1; the disease is nervous system cancer.