In non‐small‐cell lung cancer (NSCLC) with oncogenic mutations of epidermal growth factor receptor, autophagy contributed to the efficacy of TK inhibitors (TKI) by reducing tumor cell viability (Wei et al., 2013), but in RAS‐driven NSCLC, depletion of the autophagy protein Atg7 blocked tumor growth (Karsli‐Uzunbas et al., 2014). Here, EGFR is linked to neoplasm.