Instead, the autophagic flux inhibitor, chloroquine (CQ; 10 μm), enhanced the accumulation of LC3B in colorectal carcinoma cells treated with 1 μm NVP or NVP‐Iso (Fig. 6C) but decreased autophagy (Fig. S7B) and mitigated the antiproliferative effects of NVP and NVP‐Iso in these cells (Fig. S7C,D). The gene discussed is MAP1LC3B; the disease is colorectal carcinoma.