The lack of any significant correlations between the mRNA-based Ki-67-dependent Luminal A-like and Luminal B-like subtype assessment and OS/BCFI in our study is probably due to the smaller sample size in the Swiss cohort and the focus on grade 2 tumours, which is in contrast to the entirety of the IBCSG VIII and IX clinical trials. The gene discussed is MKI67; the disease is neoplasm.