LGALS3 and acute kidney injury: Importantly, transfer of WTDCsPam3CSK4 completely attenuated AKI in CDDP-injured Gal-3-/- recipients (Gal-3-/-WTDCsPam3CSK4), confirming that expression of Gal-3 on DCs was crucially responsible for DCPam3CSK4-mediated amelioration of CDDP-induced nephrotoxicity (Figure 9).