Aberrantly high expressions of miR-6875-3p in HCC tissues and cell lines potentiates tumorigenesis and differentiation metastasis via upregulating the stemness features of LCSCs both in in vitro and in vivo experiments while knockdown of miR-6875-3p inhibits stemness of LCSCs and tumorigenesis via targeting the BTG2/FAK/Akt signaling pathway [106]. The gene discussed is AKT1; the disease is hepatocellular carcinoma.