In contrast, amyloid deposits in brains of PD patients contain the 140‐residue α‐synuclein (αSyn), and T2D pancreatic amyloid deposits contain the 37‐residue islet amyloid polypeptide (IAPP).1 The amyloidogenic polypeptides exhibit distinct physiological functions: for example, Aβ is likely involved in protection of the central nervous system, αSyn regulates synaptic function, and IAPP is a neuropeptide hormone regulator of glucose homeostasis.3 This evidence concerns the gene IAPP and type 2 diabetes mellitus.