It has been demonstrated that TRM cells help to recruit immune cells to the infection site through the induction of chemokines such as CCL3 and CXCL9 in the female reproductive tract (FRT), and CCL4 in the lungs, either by direct chemokine expression or by their induction in nearby cells, such as epithelial cells [28, 29]. The gene discussed is CCL3; the disease is infection.