In addition, it has already been shown that KIR2DL2 receptor binds stronger than KIR2DL3 to HLA-C ligands, which could explain why we have the association between KIR2DL2 and LL clinical form, while KIR2DL3 associates with borderline leprosy, a less severe form of the disease [4,6]. The gene discussed is HLA-C; the disease is leprosy.