LINC00632 and hepatocellular carcinoma: Furthermore, quantitative proteomics‐based approaches have revealed the presence of CDR1as‐regulated proteins in HCC cells.69 Results from proteomic analysis and functional verification showed that overexpression of CDR1as enhanced the cell cycle progression and proliferation of HCC cells, in part, through regulation of EGFR signaling by modulating miR‐7 overexpression.