SET plays oncogenic roles in tumor formation since abnormal expression of SET led to enhanced cancer metastasis.75, 76 Interestingly, SET was highly expressed in HCC and was significantly linked to adverse clinical outcomes.77 Elsewhere, it was found that hsa_circ_0000673 enhanced HCC severity via by modulating the miR‐767‐3p/SET pathway. This evidence concerns the gene SET and hepatocellular carcinoma.