IL17A and uterine cervix neoplasm: At the 6 h time point up-regulated genes are involved in the response to lipopolysaccharide and IL-17 signaling that can indicate the activation of an inflammatory response, which is known to be highly interconnected with ER stress [21] and probably playing a pro-survival role – hyperexpression of IL-17 was shown to increase tumorigenicity of human cervical tumors in nude mice [48].