They demonstrated in subsequent experiments that overexpression of FOXA1 and HOXB13, two TFs previously shown to be associated with PC progression (Brechka et al., 2017; Yang and Yu, 2015), induced a change in the AR‐binding pattern in a benign prostate cell line such that it looked like that of PC cells (i.e., switching from predominantly N‐ARBSs to T‐ARBSs). The gene discussed is AR; the disease is pachyonychia congenita.