Because Vγ9+ subsets are functionally distinct from their Vγ9− counterparts [34], these findings suggest that Vδ1−Vδ2−Vγ9− cells but not Vδ1−Vδ2−Vγ9+ cells are related to the beneficial effects of IFN-β on MS. Here, IFNB1 is linked to myeloid sarcoma.