Since the ethanol-induced gastric ulcer is more dependent on disturbances in cytoprotective mechanisms, secretion of inflammatory mediators and antioxidant properties (De Souza et al., 2011 ▶; Repetto and Llesuy, 2002 ▶; Hernandez-Munoz et al., 2000 ▶), for studying the protective effect of BM on this gastric ulcer model, we examined some relative parameters which included total antioxidant capacity and mucosal NO content as well as serum TNF-α level. Here, TNF is linked to gastric ulcer.