We speculated that either human CD4+ T cells or human myeloid cells might be contributing to subcutaneous DLBCL growth in this model; however, neither the depletion of CD4+ T cells nor the neutralization of the myeloid cell‐specific chemokine CCL2 with specific antibodies had a discernible effect on tumor growth in the subcutaneous model (Fig EV2C and D). The gene discussed is CCL2; the disease is neoplasm.