The most prevalent genetic causes of ALS are mutations in the ‘superoxide dismutase 1’ (SOD1), ‘fused in sarcoma’ (FUS), ‘TAR DNA binding protein’ (TARDBP) gene, or a hexanucleotide repeat expansion in the ‘chromosome 9 open reading frame 72’ (C9ORF72) gene2. This evidence concerns the gene SOD1 and amyotrophic lateral sclerosis.