ULBP2 and disease arising from reactivation of latent virus: In parallel, ULBP2 mRNA was quantified showing that, while HIV-1 latent infection by itself resulted in a slight increase of ULBP2 mRNA levels, the sole exposure to Prostratin or Bryostatin led to a ULBP2 mRNA increase by 16- or 10-fold, respectively; most importantly, ULBP2 mRNA was further increased, up to 43-fold, when Prostratin, but not Bryostatin, was added to latently infected T cells.