As for SNHG14, it is also known as UBE3A‐ATS and suppresses UBE3A expression.17 SNHG14 has been found to regulate microglial activation in cerebral infarction.18 In a previous cancer study, SNHG14 was shown to be overexpressed in gastric cancer tissues and cells.19 In clear cell renal cell carcinoma, SNHG14 promotes cell migratory and invasive abilities through up‐regulation of N‐WASP protein level.20 On the other hand, SNHG14 potentiates the chemoresistance of breast cancer to trastuzumab.21 The above findings indicate that SNHG14 may be oncogenic in pancreatic cancer. This evidence concerns the gene WASL and familial pancreatic carcinoma.