A most recent study has revealed that overexpression of RAB26 can suppress LPS-induced apoptosis by inactivating the TLR4 pathway in human pulmonary microvascular endothelial cells45, suggesting that RAB26 might promote the oncogenic function of SNRPB by regulating cell apoptosis in NSCLC cells. This evidence concerns the gene RAB26 and non-small cell lung carcinoma.