INS and Hyperglycemia: The transitory hyperglycaemia is likely to be of multifactorial aetiology and may be explained by the occurrence of early phase hyperglycaemia, induced by rifampicin administration [15, 16], hyperglycaemia that normally accompanies corticosteroid administration [17], or stress hyperglycaemia ensuing from an intricate interplay between disturbed cytokine and hormone production, resulting in excessive hepatic glucose production and insulin resistance [18].