This presumption seemed to be supported by the finding that ratios of the expression of sialyltransferases (ST3GAL6 and ST6GAL1) and neuraminidase shifted towards the sialytransferases, indicating a more intensified sialylation of SLE T cell glycans, including Gal-1 binding structures. Here, ST6GAL1 is linked to systemic lupus erythematosus.