CNTN2 and infection: We conducted two inhibition studies, firstly the addition of cytochalasin B, which has been shown to inhibit HTLV-1 infection via GLUT-1 receptor interaction, rather than impacting actin microfilaments, (Manel et al., 2003), into the co-culture reduced the percentage of Tax positive HUT78 cells, i.e. reduced HTLV-1 de novo infection, by 55.9%.