FOXP3 and cancer: FOXP3 has been shown to be responsible for miR‐146a overexpression.28, 29 In addition, FOXP3 is itself regulated by PTEN and is overexpressed following PTEN knockdown.30 miRNAs are estimated to regulate a large proportion of protein coding genes in cells and therefore have a significant impact on modulating cellular pathways.25 Hence, these miRNA pathways may be activated by the key genetic alterations found in patients and targeted in murine models and function in concert to potentially aid the tumorigenic process in tubal‐derived cancer models.