Galactosemia disorders encompass three known inborn errors of galactose metabolism derived from deficiency in the enzymes galactokinase (GALK1), galactose‐1‐phosphate uridylyltransferase (GALT), and UDP‐galactose 4‐epimerase (GALE).1, 2 Classic galactosemia is an autosomal recessive disorder affecting 1 in 40 000‐60 000 births, caused by complete or partial deficiency of the GALT enzyme. Here, GALE is linked to classic galactosemia.