Bazua‐Valenti et al. recently proposed that increased calcium delivery to the DCT, as occurs following furosemide treatment, stimulates the apical calcium‐sensing receptor (CsR) to activate WNK4 and NCC, further promoting hypercalciuria (Bazúa‐Valenti et al. 2018). The gene discussed is WNK4; the disease is Hypercalciuria.