Although two Fcγ receptors particularly FcγRI (CD64) and FcγRII (CD32) have been shown to mediate ADE in phagocytic cells in vitro, FcγRIIA (CD32) was found to enhance DENV infection more efficiently than FcγRIA (CD64) [37,65,66]. The gene discussed is FCGR1A; the disease is acute disseminated encephalomyelitis.