CD36 and colorectal carcinoma: In colorectal cancer, early evidence showed that decreased stromal expression of CD36 was positively correlated with vascularization as a receptor of thrombospondin 117, and a recent work has mentioned lncRNA TINCR could inhibit proliferation and promote apoptosis by activating CD36 in CRC cells18, but the exact functional roles of CD36 in CRC development remain undefined.