In colorectal cancer, early evidence showed that decreased stromal expression of CD36 was positively correlated with vascularization as a receptor of thrombospondin 117, and a recent work has mentioned lncRNA TINCR could inhibit proliferation and promote apoptosis by activating CD36 in CRC cells18, but the exact functional roles of CD36 in CRC development remain undefined. Here, CD36 is linked to colorectal cancer.