Furthermore, we observed in p53+/+ but not in p53EE/EE or p53‐null MEFs a strong Nutlin‐inducible expression of a p53 signature including both bona fide p53 pathway genes (MSigDB hallmarks_P53_pathway) and non‐canonical targets previously identified to be critical mediators of tumor suppression (Figs 1C and EV1I) (Brady et al, 2011). The gene discussed is TP53; the disease is neoplasm.