This statement is supported by the study in CaSki and HeLa cells, showing that exogenous EGF stimulation enhances cell proliferation by activating EGFR and cyclin D1, which is independent of COX-2 levels, suggesting that the inhibitors of EGFR and cyclin D1 may be effective against cervical cancer cell proliferation. The gene discussed is EGF; the disease is cervical carcinoma.