SDF‐1‐mediated cell survival has been well characterized.5, 19 In agreement with previous studies, our in vitro and in vivo experiments showed that the cell apoptosis after MI could be attenuated by SDF‐1‐AnxA5 or SDF‐1, which may contribute to the myocardial repair. Here, ANXA5 is linked to myocardial infarction.