A recent study showed that BMSCs in a hypoxic state transferred exosomal-derived miR-193a-3p, miR-210-3p, and miR-5100 to activate signal transducer and activator of transcription 3 (STAT3) signaling in lung cancer cells and also led to an increase in the levels of vimentin and N-cadherin, two mesenchymal markers [65]. The gene discussed is STAT3; the disease is lung cancer.