The genes overexpressed in AML included well-established AML markers such as KIT, MYH11, MYC, CEBPA, MN1, MPO, SET, and HOXA10, but also genes rarely discussed in the context of AML pathogenesis, e.g., STMN1 (the most discriminative gene in our analysis), CDK6, ANGPT1 or ENO1. The gene discussed is HOXA10; the disease is acute myeloid leukemia.