The tripartite motif-containing 29 (TRIM29) is also increased in cervical cancer lesions, and its downregulation results in enhanced GSK3β expression and inhibition of the epithelial-mesenchymal transition (EMT) in cancer cells [40], which could explain how GSK3β is inactivated by Wnt/β-catenin signaling to promote tumor progression [25,41]. Here, GSK3B is linked to cervical cancer.