It is likely that this discrepancy is caused by differences in patient populations, although both had an ethnic origin, with a similar distribution of polymorphic frequencies in CD1E, but different in CD1A. On the other hand, it should be noted that the absence of association with polymorphisms of CD1 does not exclude the possibility that CD1 molecules play an important role in GBS pathogenesis. The gene discussed is CD1C; the disease is Guillain-Barre syndrome.