TNFRSF1B and neoplasm: Further, expression of lymphotoxin-A and tumor necrosis factor superfamily-14 (TNFSF14/LIGHT) by NK and complementary TNFRSF1B and TNFRSF14/HVEM (herpes virus entry mediator) by CTL may serve as a substrate for CTL–NK compartmentalization in tumors or tumor-draining lymph nodes.