In addition to CXCR6, several murine models suggest a role for CXCR3 in the migration of T cells and NK cells to the lung under steady-state levels, viral infection, and non-infectious lung-injury36–38, and antigen-specific CD8+ T cells in bronchoalveolar lavage (BAL) fluid were homogenously CXCR3hiCD49a+36. This evidence concerns the gene CD8A and viral infectious disease.