The impact on clinical outcome is also reported—AML patients who did not achieve remission when compared to, either, healthy controls, or remission patients, show increased levels in distinct glycolysis elements, such as HIF1a, hexokinase 2 (HK2), glucose transporter 1 (GLUT1), and lactate dehydrogenase (LDH) [15]. The gene discussed is SLC2A1; the disease is acute myeloid leukemia.