For example, miR‐613 acts as an upstream regulator that disturbs the interaction between YAP and WBP2, influencing the activity of the EGFR/PI3K pathway in triple‐negative BC cells.34 Similarly, cPLA2α has been shown to activate PI3k/Akt signalling to mediate the TGF‐β‐induced EMT in BC cells.36 PI3K has been linked to an extraordinarily diverse group of cellular functions via the activation of Akt. Here, AKT1 is linked to breast cancer.