In particular, polymorphisms in the thymic stromal lymphopoietin (TSLP) gene increase the risk of atopic dermatitis, food allergy, and asthma [14,15,16], whereas polymorphisms in the interleukin (IL) 33 gene predispose patients to atopic dermatitis and promote the subsequent development of asthma [17,18]. This evidence concerns the gene TSLP and atopic eczema.