An atherosclerosis‐prone mice study showed that CCL17 deficiency resulted in a reduction of atherosclerosis, which was dependent on Tregs.40, 41 Another study found that CCR4 expression was decreased by simvastatin accompanied by anti‐inflammation and lipid‐lowering effects in human endothelial cells and macrophages.42 These findings suggested CCR4 is involved in promoting atherosclerosis. The gene discussed is CCR4; the disease is atherosclerosis.