Moreover, in the present study, we demonstrated that PART1 promoted cell proliferation, migration, and invasion of NSCLC, while knockdown of PART1 performed the opposite effects on NSCLC progression.Other lncRNAs, such as MALAT1,36 HOTAIR,37 and UCA1,38 that were highly expressed in NSCLC, also promoted the progression and the inhibition of these lncRNAs suppressed the progression. The gene discussed is UCA1; the disease is non-small cell lung carcinoma.