We further measured Sam68 protein levels in skin tissue derived from Gli2‐transgenic (Gli2tg/+) mice, which spontaneously develop basaloid follicular hamartoma lesions between postnatal 45 days (P45) and P120 that then become more like nodular BCC observed in human BCC patients by around P180.20, 24 Consistently, Sam68 was upregulated in lesions in the ear skin derived from Gli2tg/+ mice, compared to the normal skin tissue derived from wild‐type (WT) mice (Figure 1C) or the adjacent normal tissue from the same tumor‐laden Gli2tg/+ mice (Figure 1D). Here, KHDRBS1 is linked to neoplasm.